Publications

A feed-forward mechanosignaling loop confers resistance to therapies targeting the MAPK pathway in BRAF-mutant melanoma.

Girard CA, Lecacheur M, Ben Jouira R, Berestjuk I, Diazzi S, Prod'homme V, Mallavialle A, Larbret F, Gesson M, Schaub S, Pisano S, Audebert S, Mari B, Gaggioli C, Leucci E, Marine JC, Deckert M, Tartare-Deckert S

NOTCH and DNA repair pathways are more frequently targeted by genomic alterations in inflammatory than in non-inflammatory breast cancers.

Bertucci F, Rypens C, Finetti P, Guille A, Adélaïde J, Monneur A, Carbuccia N, Garnier S, Dirix P, Gonçalves A, Vermeulen P, Debeb BG, Wang X, Dirix L, Ueno NT, Viens P, Cristofanilli M, Chaffanet M, Birnbaum D, Van Laere S

Special Issue: Nanobody.

Chames P, Rothbauer U

Lambert S, Borde V, Charbonnier JB, Dantzer F, Espéli O, Guirouilh-Barbat J, Llorente B, Legube G, Prioleau MN, Radicella P

Aurora B and Condensin are dispensable for chromosome arm and telomere separation during meiosis II.

Berthezene J, Reyes C, Li T, Coulon S, Bernard P, Gachet Y, Tournier S

Human RPA activates BLM’s bidirectional DNA unwinding from a nick.

Qin Z, Bi L, Hou XM, Zhang S, Zhang X, Lu Y, Li M, Modesti M, Xi XG, Sun B

EBV+ diffuse large B-cell lymphoma associated with chronic inflammation expands the spectrum of breast implant-related lymphomas.

Mescam L, Camus V, Schiano JM, Adélaïde J, Picquenot JM, Guille A, Bannier M, Ruminy P, Viailly PJ, Jardin F, Bouabdallah R, Brenot Rossi I, Bohers E, Robe C, Laurent C, Birnbaum D, Wotherspoon A, Gaulard P, Xerri L

Telomeric Transcription and Telomere Rearrangements in Quiescent Cells.

Coulon S, Vaurs M

Paternal and maternal mutations in X-STRs: A GHEP-ISFG collaborative study.

Pinto N, Pereira V, Tomas C, Loiola S, Carvalho EF, Modesti N, Maxzud M, Marcucci V, Cano H, Cicarelli R, Januario B, Bento A, Brito P, Burgos G, Paz-Cruz E, Díez-Juárez L, Vannelli S, Pontes ML, Berardi G, Furfuro S, Fernandez A, Sumita D, Bobillo C, García MG, Gusmão L

Inflammatory breast cancer cells are characterized by abrogated TGFβ1-dependent cell motility and SMAD3 activity.

Rypens C, Marsan M, Van Berckelaer C, Billiet C, Melis K, Lopez SP, van Dam P, Devi GR, Finetti P, Ueno NT, Bertucci F, Dirix P, Neven P, Vermeulen P, Dirix L, Van Laere SJ